When VZV reactivates, it spreads down the long nerve fibers (axons) that extend from sensory cell bodies to your skin.
After effects of shingles inback skin#
The neurons in the sensory ganglia have nerve fibers that supply the skin and relay information to the brain about what the body is sensing, including heat, cold, touch, pain. When you are originally exposed to VZV (chickenpox), some of the virus particles settle into nerve cells (neurons) of sensory ganglia (a group of nerve cells that connect the sensory periphery and central nervous system), where they remain for many years in an inactive, hidden (latent) form. If they get infected, they will develop chickenpox, not shingles. But if you have active shingles, contact with the fluid from your blisters can pass the virus to someone who has never had chickenpox or the chickenpox vaccine. And you cannot "catch" shingles from someone else. You cannot develop shingles unless you have had an earlier exposure to chickenpox. Fortunately, most adults who have had chickenpox never get shingles. When the virus reactivates (the cause is unknown), it is called shingles or herpes zoster virus. When the itchy red spots of childhood chickenpox disappear, the virus remains hidden in a dormant state in nerve cells, ready to strike again in later life, most often in adulthood. Shingles is caused by the varicella zoster virus (VZV)-the same virus that causes chickenpox.
Sometimes shingles can affect the eyes and cause vision loss. Most often the painful rash develops as a band or stripe on one side of the body or face and the blisters typically scab over within a week to 10 days. Shingles causes pain, burning, or a tingling sensation, along with itching and blisters. Shingles (also known as herpes zoster virus) is a neurological disorder caused by the reactivation of a viral infection in the skin's nerves.
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